Explores p53's pivotal role in cellular stability, DNA repair, and tumor suppression, emphasizing TP53 mutations and DNA double-strand break repair mechanisms.
Explores the genetic mechanisms of retinoblastoma and their clinical implications, shedding light on tumor suppression, DNA repair, and genetic recombination.
Explores personalized medicine, genetic variability, and drug response, emphasizing tailoring treatments based on individual genetic and metabolic profiles.
Explores natural selection, genetic variation, and evolutionary changes within populations, using examples like sickle cell anemia and flu virus evolution.
